Michael M. Weil, Ph.D.
Associate Professor
Phone: (970) 491-5902
Fax: (970) 491-0623
Email: Michael.Weil@colostate.edu
Office: 483 Molecular and Radiological Biosciences Building
Degrees
Ph.D., Microbiology, University of Texas at Austin
B.S., Microbiology, Louisiana State University
Research Interests
Dr. Weil's research is focused on gene mutation.
Selected Publications
Buchholz TA, Weil MM, Ashorn CL, Strom EA, Sigurdson A, Bondy M, Chakraborty R, Cox JD, McNeese MD,
Story MD. A Ser49Cys variant in the ataxia telangiectasia, mutated, gene that is more common in patients
with breast carcinoma compared with population controls. Cancer 2004;100:1345-1351.
Degg NL, Weil MM, Edwards A, Haines J, Coster M, Moody J, Ellender M, Cox R, Silver A.
Adenoma multiplicity in irradiated ApcMin mice is modified by chromosome 16 segments from BALB/c.
Cancer Res. 2003;63:2361-2363.
Haston CK, Zhou X, Gumbiner-Russo L, Gu X, Irani R, Dejournett R, Weil M, Amos C, Travis EL.
"Universal" and radiation specific loci influence murine susceptibility to radiation-induced pulmonary
fibrosis. Cancer Res. 2002;62:3782-3788.
Haston CK, Wang M, Dejournett R, Zhou X, Ni D, Gu X, King TM, Weil M, Newman RA, Amos C, Travis EL.
Bleomycin hydrolase and a genetic locus within the MHC affect risk for pulmonary fibrosis in mice.
Human Mol. Genet. 2002;11:1855-1863.
Yu YE, Zhang Y, Unni E, Shirley CR, Deng JM, Russell LD, Weil MM, Behringer RR, Meistrich ML.
Abnormal spermatogenesis and reduced fertility in transition nuclear protein l-deficient mice.
Proc. Natl. Acad. Sci. USA 2002;97:4683-4688.
Bonnen PE, Story MD, Ashorn CL, Buchholz TA, Weil MM, Nelson DL. Haplotypes at ATM identify coding
sequence variation and indicate a region of reduced recombination. Am. J. Hum. Genet. 2002;67:1437-1451.
Weil MM, Xia C, Xia X, Gu X, Amos CI, Mason KA. A chromosome 15 QTL controls levels of
radiation-induced jejunal crypt cell apoptosis in mice. Genomics 2001;72:73-77.
Yu Y, Okayasu R, Weil MM, Silver A, McCarthy M, Zabriskie R, Cox R, Ullrich RL. Prkdc polymorphism,
genomic instability and radiation response in BALB/c mice. Cancer Res. 2001;61:1820-1824.
Weil MM, Kittrell FS, Yu Y, McCarthy M, Zabriskie RC, Ullrich RL. Radiation induces genomic
instability and ductal dysplasia in Atm heterozygous mice. Oncogene 2001;20:4409-4411.
Wu Y, Pan S, Che S, He G, Nelman-Gonzalez M, Weil MM, Kuang J. Overexpression of Hp95 induces G1
phase arrest in confluent HeLa cells. Differentiation 2001;67:139-153.
Zhao M, Shirley CR, Yu YE, Mohapatra B, Zhang Y, Unni E, Deng JM, Arango NA, Terry NHA, Weil MM,
Russell LD, Behringer RR, Meistrich ML. Targeted disruption of the transition protein 2 gene affects
sperm chromatin structure and reduces fertility in mice. Mol. Cell. Biol. 2001;21:7243-7255.
Story M, Weil M, Buchholz T, Cox J, Strom E. ATM heterozygosity in the normal tissue response to
Radiotherapy. Radiat. Res. Volume 2. Congress Proceedings 1999;630-633.
Mailing Address
Environmental & Radiological Health Sciences
1681Campus Delivery
Colorado State University
Fort Collins, CO 80523
Phone: (970) 491-7038
Fax: (970) 491-2940
Email:
ERHSDepartment
